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Hey there gorgeous.

you have stumbled upon the secret diary of jiali :D

welcome.

29th May, 2009
Dear Diary,

i went to the singapore flyer on a friday and that is why this notebook you've looking at now has a post-it-note stuck to it saying 'flyer friday'. i think alliterations are awesome, dont you think so too? i hope you do, 'cause if not i'll attempt to crush you with my little pinky. it tells me to remember the sweet times i've had with a guy called zhnqn. that's his name with the vowels stashed out. i hope i can remember his name and his face and his shoulder and his big big palm that i used to hold, should i turn old and get stricken by dementia someday.

it sounds creepy to be old, doesn't it? D:
we ate yummy red strawberries coated with dark chocolate as adults call 'fondue'. i think they really nice and i like them alot. mayb someday i'll open a chocolatier and i'll name it zhnqn. people wont know how to pronounce it and that's gnna be the cool thing about it :D

zHnQn. ZhNqN. ZHNQN. zhnqn.
sounds good any way, doesnt it! :D

k tht's th end for today i hope i wont get dementia when i grow old.
byebye.

xoxo,
♥ jiali

♥ ♥ ♥ ♥ ♥ ♥

g6pd.

Wednesday, November 05, 2008 @ 3:51 PM

G6PD Deficiency is a hereditary abnormality in the activity of an erythrocyte (red blood cell) enzyme. This enzyme, glucose-6-phosphate dehydrogenase (G-6-PD), is essential for assuring a normal life span for red blood cells, and for oxidizing processes. This enzyme deficiency may provoke the sudden destruction of red blood cells and lead to hemolytic anemia with jaundice following the intake of fava beans, certain legumes and various drugs (see a complete list of drugs and foodstuffs to avoid).

The defect is sex-linked, transmitted from mother (usually a healthy carrier) to son (or daughter, who would be a healthy carrier too; see a diagram of inheritance probabilities). This is due to the fact that the structure of G-6-PD is carried on the X chromosome: As stated by Ernest Beutler, M.D., "in females, only one of the two X chromosomes in each cell is active; consequently, female heterozygotes for G-6-PD deficiency have two populations of red cells; deficient cells and normal cells."

If your child is exposed to a medication or infection that causes oxidative stress in his blood cells, sometimes there are no symptoms at all. In more serious cases, your child may exhibit symptoms of anemia, including pallor, fatigue, rapid heartbeat, rapid breathing or shortness of breath, jaundice( yellowing of the skin and eyes), an enlarged spleen, or dark, tea-colored urine . Once the trigger is removed or resolved, the symptoms of G6PD deficiency usually disappear fairly quickly, typically within a few weeks. Newborns may develop persistent and more serious jaundice as a result of G6PD.

Miscellaneous elements besides drugs that should be avoided
Fava Beans
(Some, but only some, prefer also to avoid red wine, all legumes, blueberries [also yogurts containing these], soya products, tonic water).

Chemicals

Moth Balls (napthalene)
Methylene blue

~~no wonder i could not do that experiment.~~

shizzz.